[1]傅大放,赵联芳.Cr6+对厌氧微生物的毒性作用研究[J].东南大学学报(自然科学版),2002,32(4):672-675.[doi:10.3969/j.issn.1001-0505.2002.04.031]
 Fu Dafang,Zhao Lianfang.Study on the toxicity to anaerobic microbe by Cr6+[J].Journal of Southeast University (Natural Science Edition),2002,32(4):672-675.[doi:10.3969/j.issn.1001-0505.2002.04.031]
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Cr6+对厌氧微生物的毒性作用研究()
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《东南大学学报(自然科学版)》[ISSN:1001-0505/CN:32-1178/N]

卷:
32
期数:
2002年第4期
页码:
672-675
栏目:
环境科学与工程
出版日期:
2002-07-20

文章信息/Info

Title:
Study on the toxicity to anaerobic microbe by Cr6+
作者:
傅大放 赵联芳
东南大学环境工程系, 南京 210096
Author(s):
Fu Dafang Zhao Lianfang
Department of Environmental Engineering, Southeast University, Nanjing 210096, China
关键词:
Cr6+质量浓度 抑制作用 厌氧
Keywords:
Cr6+concentration inhibition anaerobic
分类号:
X506
DOI:
10.3969/j.issn.1001-0505.2002.04.031
摘要:
通过对取自ASBR中的厌氧污泥Cr6+中毒的产甲烷活性恢复试验,研究了不同Cr6+质量浓度对产甲烷微生物的抑制程度及抑制作用的分子机理,得出结论:当Cr6+质量浓度小于30 mg/L时,是生理毒素; 当Cr6+质量浓度大于110 mg/L时,是杀菌性毒素以及当Cr6+质量浓度小于10 mg/L时,其抑制作用发生在反应后期; 当Cr6+质量浓度大于30 mg/L时,其抑制作用发生在反应初期.通过对不同Cr6+质量浓度下的厌氧微生物群落及产甲烷菌形态的照片分析,发现Cr6+对厌氧微生物群落的组成和厌氧微生物的形态没有影响,但是高质量浓度Cr6+强烈抑制了产甲烷菌的活性.
Abstract:
Based on the experiment of the toxicity to the anaerobic sludge methanogens and the recovery of the methanogentic activity, the level of inhibition to methanogens by various Cr6+ concentrations and the molecule mechanism of inhibition are analyzed. It is concluded that when the Cr6+concentration is less than 30 mg/L, it is physiological toxin, but bactericidal toxin when concentration is more than 110 mg/L. The inhibition takes place in anaphase when Cr6+concentration is less than 10 mg/L, and in prophase when Cr6+ concentration is more than 30 mg/L. Based on the photograph discussion of the anaerobic species and the methanogens morphology under the various Cr6+concentrations, it is noted that the Cr6+ inhibition has no obvious influence on the anaerobic species and the methanogens morphology, but it strongly inhibits the activity of methanogens.

参考文献/References:

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备注/Memo

备注/Memo:
作者简介: 傅大放(1966—),男,博士生, 副教授,fudafang@public1.ptt.js.cn.
更新日期/Last Update: 2002-07-20